Gain Therapeutics (GANX) announced a poster presentation was made at the Society for Neuroscience annual meeting, Neuroscience 2025, held November 15th-19th in San Diego, CA. The poster outlined new evidence from three preclinical models supporting the Company’s lead drug candidate’s, GT-02287, proposed mechanism of action of facilitating trafficking of glucocerebrosidase to the mitochondria as well as the lysosomes, supporting its potential as a disease-modifying therapy for Parkinson’s disease. Data shows: GT-02287 reduces mitochondrial stress and enhances neuronal survival in MPP+ treated rat dopaminergic neurons; GT-02287 decreases staining for MIRO1, suggesting improved mitochondrial health in alpha-synuclein-induced mouse PD model ; GT-02287 improves mitochondrial function by facilitating GCase trafficking to the mitochondria in patient-derived fibroblasts harboring GBA1 mutation
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